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Stasis
Versus Dynamism
by Doug
McGuff, MD, ©1999
Recently
I attended a lecture and book signing by Virginia Postrel. Ms. Postrel
is the editor of Reason magazine and author of the new book The Future
and Its Enemies. The subject of Ms. Postrel's book and lecture was the
concept of stasis versus dynamism. In her introduction Ms. Postrel states
"Stasists and dynamists disagree about the limits and use of knowledge.
Stasists demand that knowledge be articulated and easily shared. Dynamists,
by contrast, appreciate dispersed, often tacit knowledge...Those conflicts
lead to very different beliefs about good institutions and rules: Stasists
seek specifics to govern each new situation and keep things under control.
Dynamists want to limit universal rule making to broadly applicable and
rarely changed principles, within which people can create and test countless
combinations." Having heard the above definition, I was embarrassed
to realize my own stasist tendencies. Stasist tendencies tend to be highly
represented in the H.I.T. and SuperSlow communities, as can be easily
seen in much of my own writing. What I came to realize as I listened to
this lecture is that every improvement I have ever brought to my own training
came from breaking away from my stasist tendencies and incorporating dynamist
thinking and problem solving.
What follows
are some representations of how we have broken out of stasis at Ultimate
Exercise in the past and some of the experiments we are currently involved
in. As I present these to you I must offer a few disclaimers... First,
while these investigations are controlled experiments they are not research.
Research implies randomization, removal of bias and statistical control
which we are not able to incorporate at Ultimate Exercise. Second, the
theories we are testing are not endorsed by Ultimate Exercise (except
where noted), the SuperSlow Exercise Guild or any other person or certifying
body. Finally, much of what we are trying is unproven and may not work.
We are not advocating that everything we are trying will work, but if
it seems to, we will make it known in future articles.
Stasis
in H.I.T.
In Chapter
1 of the SuperSlow Technical Manual Ken Hutchins describes some of his
early training with Jim Flanagan... "I continuously trained with
Jim Flanagan for eight months-from August 1979 to April 1980...Eventually,
Jim knew my exact resistance requirements for each Nautilus exercise.
I, likewise, knew his and remember some of them to this day". How
was it that many years later Ken could recall Jim's resistance requirements
so well? I believe that, like so many of us in H.I.T., the resistance
never changed over that eight month period. I remember doing the same
Arthur Jones prescribed routine every Monday, Wednesday and Friday from
1979-1989 with no change in resistance. I can still recall the exact weight
on every movement in that routine. I always used a rep range of 8-12,
but because I never quite made that 12th rep, I never progressed the weight.
I assume a similar pattern occurred with Ken and Jim until Ken broke stasis
with the SuperSlow protocol, producing rapid results for the first time
in months. Like many Nautilus enthusiasts of the day, I rationalized that
since intensity was the key stimulus, it was acceptable that my weights
not progress...after all, I was training to failure and beyond. Talk about
Stasis! I was using my stasist beliefs as a shield against the undeniable
fact that if your weights aren't progressing, then you aren't getting
stronger.
Training
as a Dynamic Process
I am finding
one of the major flaws in our attempts to understand the nature of the
weight training stimulus is the assumption that there must be a single
key stimulus that underlies the stimulus-organism-response relationship.
At Ultimate Exercise, Terry Carter and I are finding that the stimulus
is actually multi-factorial and that the relative contribution of each
stimulus changes as a subject progresses. What was once a positive stimulus
can become negative in the changing context of progress. What does seem
to be a constant however, is that for strength increases to occur, resistance
must progress over time. While this seems obvious and straightforward,
there are several barriers to weight progression which occur at different
stages of training. To understand these barriers we need to discuss the
contribution of the various stimuli. The stimuli we are currently focusing
on are inroad, metabolic effect, and exposure to resistance.
Inroad (or
the momentary weakening of a muscle) is a widely recognized stimulus for
muscle growth and is a cornerstone of H.I.T. philosophy. Our own views
on inroad can be found in previous articles and in Ultimate Exercise:Bulletin
1. Inroad is not the only stimulus for strength progression; if it were,
then "hyper" training would produce rapid, uninterrupted progress.
"Hyper" refers to training to failure with manual resistance
added to the negative portion of every rep and then rep assist on the
positives after failure with continued application of extra negative resistance
manually. This is carried to the point where the subject can no longer
even move the affected limb. If inroad were the only issue, this sort
of workout performed regularly should produce amazing progress. Unfortunately,
for those of us who tried it, it simply resulted in a rapid stalemate
and profound fatigue. It appears that "enough" inroad is a requirement,
but like other aspects of training more is not necessarily better. Inroad
appears to be a major contributor in the early stages of training because
it is hard for the novice to achieve, and it is rare for the beginner/intermediate
subject to have the capability of invoking too much of it. In more advanced
stages, a subject can invoke a degree of inroad that is difficult to recover
from; indeed, one of the beneficial side effects of weight progression
is to protect against excessive inroad. However, even with weight progression,
the degree of motor unit recruitment that a subject can invoke can produce
excessive inroad fairly easily. We are finding better rates of progress
by limiting depth of inroad as subjects get stronger. We are even experimenting
with stopping our more advanced subjects short of failure (we will discuss
this in more detail later).
Another key
stimulus in the growth process seems to be the "metabolic effect"
of the workout: the sensation of heavy breathing, rapid heart rate, tunnel
vision, roaring in the ears, nausea etc. These are all sensations of your
body trying to supply nutrients to the working muscles and remove waste
products and buffer metabolic acidosis. This condition seems to offer
the milieu in which the growth process is most easily triggered and seems
to be involved with the "indirect effect" on the non-working
muscles. While no one fully understands the relationship between mechanical
work and metabolic work, we can demonstrate that increased strength without
a concomitant decrease in volume quickly leads to what we call "metabolic
meltdown". At this point the subject requires extended "carpet
time" and attempts at additional exercise require large decreases
in resistance to perform at a reasonable TUL. Pushing a subject beyond
this point quickly stalls progress regardless of the recovery interval.
So, metabolic effect is also a component of the growth stimulus but again,
more is not better. As a subject becomes stronger "metabolic meltdown"
occurs sooner and sooner in the workout.
The final
contribution to the growth stimulus that I will mention is what we call
"exposure to the weight". It seems that exposure to increasing
resistance is one of the most important stimuli in the growth process.
At the cellular level, satellite cells (myogenic stem cells) seem to respond
to weight exposure by upregulating their receptors for IGF-1 (Insulin-like
growth factor-1) which will bind circulating IGF-1 and result in growth.
Indeed, without weight progression the other components do not provide
a stimulus for growth. Some degree of inroad and metabolic effect seem
to augment the effect of weight progression, particularly in early and
intermediate stages of training. It has been demonstrated that intensity
and metabolic effect are tied to Growth hormone release (precursor of
IGF-1). We can see that increased IGF-1 is integral to the growth process,
but unless IGF-1 receptors are upregulated by exposure to heavier weight,
the existing receptors will become saturated and all the intensity in
the world and all the IGF-1 in the world will not make a difference. (Please
understand that this statement is my own conjecture). However, as a subject
gets stronger, he develops the capability of producing much larger amounts
of inroad and subsequently much larger metabolic effects (note that I
am not claiming larger percentages of inroad, just larger amounts...i.e.
25% of 400lbs is a lot more inroad than 25% of 200lbs). In the early stages
of training the degree of inroad and metabolic effect seems to augment
weight progression and strength gains. In the intermediate stages and
early advanced levels inroad and metabolic effect appears to be permissive,
not inhibiting or augmenting strength gain but still desirable because
of the metabolic conditioning. In truly advanced stages, inroad and metabolic
effect seems to become "toxic"; that is, the effect is so severe
that it seems to prevent continued weight progression. I speculate that
this may have to do with release of cortisol and other stress hormones
that down-regulate IGF-1 receptors. The biological message seems to be
"this conflict is so severe that rather than allowing further adaptations
to this environment, it would be better to leave this environment"
or something to that effect. We are finding at this point that protocol
needs to be modified. Inroad and metabolic effect need to be controlled
so that weight progression can continue. In the articles that follow we
will describe the modifications we make as clients progress and how we
manipulate the dynamic factors involved. 
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